Title
Transcriptional regulation of the rat glutamine synthetase gene by tumor necrosis factor-alpha
Abbreviated Journal Title
Eur. J. Biochem.
Keywords
glutamine synthetase; transcription; tumor-necrosis factor; SENSITIVE METHOD; CELLS; PROTEINS; PHOSPHORYLATION; EXPRESSION; CYTOKINES; ENHANCER; BINDING; ENZYME; ASSAY; Biochemistry & Molecular Biology
Abstract
The expression of glutamine synthetase (GS) is induced in rat skeletal muscle cells (L-6) in response to treatment with the inflammatory cytokine tumor necrosis factor alpha (TNF-alpha). This paper reports the regulation of GS expression in rat skeletal muscle which expresses high levels of GS. TNF-alpha treatment leads to a 3-4-fold increase in GS activity in a dose-dependent and time-dependent manner. Northern-blot analysis of GS mRNA revealed an increased mRNA concentration, reaching a peak at 12 h in response to TNF-alpha treatment. To monitor transcriptional activation of GS by TNF-alpha, and to identify a TNF-alpha-responsive element in the GS promoter, L6 cells were treated with TNF-alpha following transfection of GS-chloramphenicol-acetyltransferase (CAT) constructs. The first 251-bp fragment at the GS upstream sequence showed basal promoter activity, but failed to show any TNF-alpha-inducible activity. However, a 2.5-3-fold induction was noted in constructs extending up to 1.1 kb. This data demonstrates that the rat GS gene is transcriptionally regulated by TNF-alpha and identifies a TNF-alpha-responsive region at the 5' flanking sequence of the GS gene.
Journal Title
European Journal of Biochemistry
Volume
254
Issue/Number
1
Publication Date
1-1-1998
Document Type
Article
Language
English
First Page
70
Last Page
74
WOS Identifier
ISSN
0014-2956
Recommended Citation
"Transcriptional regulation of the rat glutamine synthetase gene by tumor necrosis factor-alpha" (1998). Faculty Bibliography 1990s. 2202.
https://stars.library.ucf.edu/facultybib1990/2202
Comments
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