Title

The Collapsin Response Mediator Protein 5 Onconeural Protein Is Expressed in Schwann Cells Under Axonal Signals and Regulates Axon-Schwann Cell Interactions

Authors

Authors

J. P. Camdessanche; K. Ferraud; N. Boutahar; F. Lassabliere; M. Mutter; M. Touret; P. Kolattukudy; J. Honnorat;J. C. Antoine

Comments

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Abbreviated Journal Title

J. Neuropathol. Exp. Neurol.

Keywords

CRMP5; Paraneoplastic neurologic syndromes; Peripheral neuropathy; Schwann cells; GROWTH-CONE COLLAPSE; MOLECULAR CHARACTERIZATION; DIFFERENTIAL; EXPRESSION; NEURITE OUTGROWTH; PERIPHERAL-NERVE; MOUSE-BRAIN; RHO-KINASE; MYELINATION; NEURONS; SYSTEM; Clinical Neurology; Neurosciences; Pathology

Abstract

Collapsin response mediator protein 5 (CRMP5) is one of the rare peripheral nerve antigens that is a target of autoantibodies in a paraneoplastic peripheral neuropathy. The pattern of axonal and myelin alterations suggests that CRMP5 is involved in axon-Schwann cell interaction. We examined CRMP5 expression and function in primary cultures of Schwann cells and neurons and at various developmental and regenerating stages of rat sciatic nerve and in CRMP5-deficient mice in vivo. Collapsin response mediator protein 5 was strongly expressed during postnatal development and regeneration and decreased with myelination. It was mainly expressed by immature Schwann cells and persisted in Remak cells in the adult; however, a subpopulation of Schwann cells that were induced to myelinate also expressed CRMP5. We identified 2 axonal molecular cues regulating CRMP5 expression: human neuregulin type 1, which induces CRMP5 expression in immature and premyelinating Schwann cells, and cyclic adenosine monophosphate, which inhibits CRMP5 expression when Schwann cells begin myelination. Collapsin response mediator protein 5-deficient mice showed abnormal Schwann process extension resulting in abnormal cell-axon segregation, indicating that CRMP5 is involved in the morphologic adaptation of Schwann cells to surround axons. These results demonstrate the importance of CRMP5 in axon-Schwann cell cooperation during development and regeneration.

Journal Title

Journal of Neuropathology and Experimental Neurology

Volume

71

Issue/Number

4

Publication Date

1-1-2012

Document Type

Article

Language

English

First Page

298

Last Page

311

WOS Identifier

WOS:000302178700004

ISSN

0022-3069

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