A Single Mutation Unlocks Cascading Exaptations In The Origin Of A Potent Pitviper Neurotoxin
Keywords
evolutionary biophysics; evolutionary innovation; exaptation; gene duplication; venom
Abstract
Evolutionary innovations and complex phenotypes seemingly require an improbable amount of genetic change to evolve. Rattlesnakes display two dramatically different venom phenotypes. Type I venoms are hemorrhagic with low systemic toxicity and high expression of tissue-destroying snake venom metalloproteinases. Type II venoms are highly neurotoxic and lack snake venom metalloproteinase expression and associated hemorrhagic activity. This dichotomy hinges on Mojave toxin (MTx), a phospholipase A 2 (PLA2) based β-neurotoxin expressed in Type II venoms. MTx is comprised of a nontoxic acidic subunit that undergoes extensive proteolytic processing and allosterically regulates activity of a neurotoxic basic subunit. Evolution of the acidic subunit presents an evolutionary challenge because the need for high expression of a nontoxic venom component and the proteolytic machinery required for processing suggests genetic changes of seemingly little immediate benefit to fitness. We showed that MTx evolved through a cascading series of exaptations unlocked by a single nucleotide change. The evolution of one new cleavage site in the acidic subunit unmasked buried cleavage sites already present in ancestral PLA2s, enabling proteolytic processing. Snake venom serine proteases, already present in the venom to disrupt prey hemostasis, possess the requisite specificities for MTx acidic subunit proteolysis. The dimerization interface between MTx subunits evolved by exploiting a latent, but masked, hydrophobic interaction between ancestral PLA2s. The evolution of MTx through exaptation of existing functional and structural features suggests complex phenotypes that depend on evolutionary innovations can arise from minimal genetic change enabled by prior evolution.
Publication Date
4-1-2018
Publication Title
Molecular Biology and Evolution
Volume
35
Issue
4
Number of Pages
887-898
Document Type
Article
Personal Identifier
scopus
DOI Link
https://doi.org/10.1093/molbev/msx334
Copyright Status
Unknown
Socpus ID
85044602078 (Scopus)
Source API URL
https://api.elsevier.com/content/abstract/scopus_id/85044602078
STARS Citation
Whittington, A. Carl; Mason, Andrew J.; and Rokyta, Darin R., "A Single Mutation Unlocks Cascading Exaptations In The Origin Of A Potent Pitviper Neurotoxin" (2018). Scopus Export 2015-2019. 10400.
https://stars.library.ucf.edu/scopus2015/10400