Avascular Necrosis Of The Hips With Increased Activity On 68Ga-Dotatate Pet/Ct
Abstract
Prolonged exposure to cortisol is one of the major causes of avascular bone necrosis (AVN). We report on a case of a woman with Cushing syndrome attributed to ectopic adrenocorticotropic hormone-secreting tumor who was evaluated with whole-body PET/CT study using Ga-DOTATATE. The scan showed increased activity by both femoral heads, corresponding to the margins of bilateral AVN seen on MRI. The presented data suggests AVN-induced reactive inflammatory alterations adjacent to the necrotic segment of the bone, which can be effectively targeted using radiolabeled somatostatin (SST) analogs.
Publication Date
3-1-2017
Publication Title
Clinical nuclear medicine
Volume
42
Issue
3
Number of Pages
214-215
Document Type
Article
Personal Identifier
scopus
DOI Link
https://doi.org/10.1097/RLU.0000000000001513
Copyright Status
Unknown
Socpus ID
85028240024 (Scopus)
Source API URL
https://api.elsevier.com/content/abstract/scopus_id/85028240024
STARS Citation
Papadakis, Georgios Z.; Millo, Corina; Karantanas, Apostolos H.; Bagci, Ulas; and Patronas, Nicholas J., "Avascular Necrosis Of The Hips With Increased Activity On 68Ga-Dotatate Pet/Ct" (2017). Scopus Export 2015-2019. 5505.
https://stars.library.ucf.edu/scopus2015/5505